Your Atrial Fibrillation Queries Answered4344388
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In AF, the heart's standard electrical "pacemaker" is bombarded by competing electrical charges which originate inside the atria. These competing impulses initiate chaotic muscle contractions, so rather than contracting forcefully to move the blood out, the atria quiver or "fibrillate." This could result in a loss of up to 30 % from the "atrial kick" that contributes towards the blood output from the heart, and may send heart prices up to 230 to 250 beats per minute.
Blood thinning agents, or anticoagulants, like aspirin and Coumadin enable prevent clots from forming inside the fibrillating atria, and shield against stroke. Cardiologists disagree about when to provide anticoagulation therapy, but most patients with chronic AF require it.
The important danger from anticoagulants includes possible bleeding or hemorrhage. Anticoagulants slow blood clotting time, but thinning the blood too much can carry risks. Inside the elderly, overly prolonged clotting instances can enhance the opportunity of internal bleeding. If you play speak to sports or are traumatized by an injury, "thinned blood" which clots gradually can be risky.
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Lately, much more physicians are comfortable with leaving medically stable individuals in AF, and providing them with "rate control" and anticoagulation therapy. The objective of rate handle will be to give the least toxic drug within the lowest powerful dose. In this way we can handle the heart rate even though safeguarding the patient against stroke with anticoagulants.
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